By John M. Weiler
This detailed source offers allergic and airway stipulations noticeable in leisure and aggressive athletes-covering themes from exercise-induced bronchial asthma to chilly air-induced rhinitis.
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Extra resources for Allergic and Respiratory Disease in Sports Medicine (Clinical Allergy and Immunology)
D. Department of Medicine, University of Wisconsin—Madison, and William S. Middleton Memorial Veterans Hospital, Madison, Wisconsin William W. D. Department of Medicine, University Hospitals—CSC, University of Wisconsin Medical School, Madison, Wisconsin Ernest N. D. Brenham Clinic, Brenham, Texas Christodoulos S. D. Division of Allergy/Immunology, Department of Internal Medicine, University of Texas Health Science Center at San Antonio, San Antonio, Texas Robert E. D. D. Department of Respiratory Medicine, Royal Prince Alfred Hospital, Sydney, Australia Joseph D.
Neither this book nor any part may be reproduced or transmitted in any form or by any means, electronic or mechanical, including photocopying, microfilming, and recording, or by any information storage and retrieval system, without permission in writing from the publisher. MARCEL DEKKER, INC. com Current printing (last digit): 10 9 8 7 6 5 4 3 2 1 PRINTED IN THE UNITED STATES OF AMERICA Page iii To Kay, Rebecca, and Jamie and to the Comets Page iv This page intentionally left blank. Page v Preface Studies and reports presented over the past two decades provide the basis for recognizing the importance of the disorders described in this text.
However, the time rate of change of PO2 and PCO2 in expired gas also reflect continued CO2 output and O2 uptake. Thus, with higher and of exercise, the expired gas waveforms become steeper. The end-expired PCO2 (PETCO2) usually exceeds arterial PaCO2 in heavy exercise [64,118]. If PETCO2 remains below PaCO2 at moderate to heavy work intensities, there may be larger than normal contribution to expired gas from acini with high /Q ratios, such as might occur in pulmonary vascular or parenchymal disorders.